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primary antibodies against aoc1  (Proteintech)


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    Proteintech primary antibodies against aoc1
    <t>AOC1</t> is highly expressed in gastric cancer tissues compared with normal tissues. ( A ) The red and gray boxes indicate gastric cancer and normal tissues, respectively. Data were obtained from the GEPIA website, including 408 gastric cancer samples and 211 controls. The y-axis indicated the log2-transformed gene expression levels. qPCR ( B ) and western blot ( C ) were performed to detect AOC1 expression in the tumor and paracancerous tissues of 30 patients with gastric cancerthe. *P<0.05; **P<0.01; ***P<0.001.
    Primary Antibodies Against Aoc1, supplied by Proteintech, used in various techniques. Bioz Stars score: 92/100, based on 4 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/primary antibodies against aoc1/product/Proteintech
    Average 92 stars, based on 4 article reviews
    primary antibodies against aoc1 - by Bioz Stars, 2026-03
    92/100 stars

    Images

    1) Product Images from "AOC1 Contributes to Tumor Progression by Promoting the AKT and EMT Pathways in Gastric Cancer"

    Article Title: AOC1 Contributes to Tumor Progression by Promoting the AKT and EMT Pathways in Gastric Cancer

    Journal: Cancer Management and Research

    doi: 10.2147/CMAR.S225229

    AOC1 is highly expressed in gastric cancer tissues compared with normal tissues. ( A ) The red and gray boxes indicate gastric cancer and normal tissues, respectively. Data were obtained from the GEPIA website, including 408 gastric cancer samples and 211 controls. The y-axis indicated the log2-transformed gene expression levels. qPCR ( B ) and western blot ( C ) were performed to detect AOC1 expression in the tumor and paracancerous tissues of 30 patients with gastric cancerthe. *P<0.05; **P<0.01; ***P<0.001.
    Figure Legend Snippet: AOC1 is highly expressed in gastric cancer tissues compared with normal tissues. ( A ) The red and gray boxes indicate gastric cancer and normal tissues, respectively. Data were obtained from the GEPIA website, including 408 gastric cancer samples and 211 controls. The y-axis indicated the log2-transformed gene expression levels. qPCR ( B ) and western blot ( C ) were performed to detect AOC1 expression in the tumor and paracancerous tissues of 30 patients with gastric cancerthe. *P<0.05; **P<0.01; ***P<0.001.

    Techniques Used: Transformation Assay, Gene Expression, Western Blot, Expressing

    AOC1 expression is effectively knocked down by siRNA transfection in human gastric cancer cells. qRT-PCR assay was used to detect the interference efficiencies of siRNAs targeting AOC1 on mRNA levels in human ( A ) AGS and ( B ) MKN45 cells. ( C ) Western blot validated the effectiveness of AOC1 knockdown on protein levels. A scrambled siRNA was used as the negative control (NC). All experiments were performed in triplicate, independently. *P<0.05.
    Figure Legend Snippet: AOC1 expression is effectively knocked down by siRNA transfection in human gastric cancer cells. qRT-PCR assay was used to detect the interference efficiencies of siRNAs targeting AOC1 on mRNA levels in human ( A ) AGS and ( B ) MKN45 cells. ( C ) Western blot validated the effectiveness of AOC1 knockdown on protein levels. A scrambled siRNA was used as the negative control (NC). All experiments were performed in triplicate, independently. *P<0.05.

    Techniques Used: Expressing, Transfection, Quantitative RT-PCR, Western Blot, Knockdown, Negative Control

    AOC1 knockdown induces growth inhibition in human gastric cancer cells. ( A ) and ( B ) After transfection with siNC or si-AOC1, the viability of AGS and MKN45 cells was detected by using CCK-8 assay. ( C ) and ( D ) Clone formation ability of AOC1 silenced gastric cancer cells was detected. All experiments were performed in triplicate. *P<0.05.
    Figure Legend Snippet: AOC1 knockdown induces growth inhibition in human gastric cancer cells. ( A ) and ( B ) After transfection with siNC or si-AOC1, the viability of AGS and MKN45 cells was detected by using CCK-8 assay. ( C ) and ( D ) Clone formation ability of AOC1 silenced gastric cancer cells was detected. All experiments were performed in triplicate. *P<0.05.

    Techniques Used: Knockdown, Inhibition, Transfection, CCK-8 Assay

    Knockdown of AOC1 inhibits cell invasion and migration in human gastric cancer cells. ( A ) and ( C ) Transwell assays detecting the invasion and migration of human AGS and MKN45 cells. ( B ) and ( D ) Invaded and migrated cells in si-AOC1 group or NC group were counted. All experiments were performed for three repeated times. *P<0.05.
    Figure Legend Snippet: Knockdown of AOC1 inhibits cell invasion and migration in human gastric cancer cells. ( A ) and ( C ) Transwell assays detecting the invasion and migration of human AGS and MKN45 cells. ( B ) and ( D ) Invaded and migrated cells in si-AOC1 group or NC group were counted. All experiments were performed for three repeated times. *P<0.05.

    Techniques Used: Knockdown, Migration

    Knockdown of AOC1 induces apoptosis in human gastric cancer cells. ( A ) and ( B ) The effect of AOC1 knockdown on the apoptosis of AGS cells was detected using flow cytometry. ( C ) and ( D ) The effect of AOC1 knockdown on the apoptosis of MKN45 cells was detected using flow cytometry. All experiments were performed in triplicate. *P<0.05.
    Figure Legend Snippet: Knockdown of AOC1 induces apoptosis in human gastric cancer cells. ( A ) and ( B ) The effect of AOC1 knockdown on the apoptosis of AGS cells was detected using flow cytometry. ( C ) and ( D ) The effect of AOC1 knockdown on the apoptosis of MKN45 cells was detected using flow cytometry. All experiments were performed in triplicate. *P<0.05.

    Techniques Used: Knockdown, Flow Cytometry

    Knockdown of AOC1 induces activation of the mitochondrial apoptosis pathway, and also inhibits the AKT signaling pathway and EMT process. ( A and B ) The key members of the mitochondrial apoptosis pathway, including Bax, Bcl2, Caspase-9, and Caspase-3, were detected by Western blot. ( C and D ) AKT signaling pathway members, including AKT, p-AKT, Cyclin D1, and p70S6K, were detected by Western blot. ( E and F ) EMT-related proteins, including E-cadherin, N-cadherin, SNAIL and Slug, were detected by Western blot. All experiments were performed in triplicate. *P<0.05.
    Figure Legend Snippet: Knockdown of AOC1 induces activation of the mitochondrial apoptosis pathway, and also inhibits the AKT signaling pathway and EMT process. ( A and B ) The key members of the mitochondrial apoptosis pathway, including Bax, Bcl2, Caspase-9, and Caspase-3, were detected by Western blot. ( C and D ) AKT signaling pathway members, including AKT, p-AKT, Cyclin D1, and p70S6K, were detected by Western blot. ( E and F ) EMT-related proteins, including E-cadherin, N-cadherin, SNAIL and Slug, were detected by Western blot. All experiments were performed in triplicate. *P<0.05.

    Techniques Used: Knockdown, Activation Assay, Western Blot

    IGF-1 blocked the inhibition of cell proliferation, migration and invasion caused by AOC1 knockdown. AOC1 low expressing cells were treated with IGF-1, an agonist of the AKT pathway. ( A ) CCK8 was performed to detected the proliferation of each group cells. ( B ) Transwell was performed to detected the migration and invasion of each group cells. *P<0.05 vs. NC group; #P<0.05 vs. si-AOC1 group.
    Figure Legend Snippet: IGF-1 blocked the inhibition of cell proliferation, migration and invasion caused by AOC1 knockdown. AOC1 low expressing cells were treated with IGF-1, an agonist of the AKT pathway. ( A ) CCK8 was performed to detected the proliferation of each group cells. ( B ) Transwell was performed to detected the migration and invasion of each group cells. *P<0.05 vs. NC group; #P<0.05 vs. si-AOC1 group.

    Techniques Used: Inhibition, Migration, Knockdown, Expressing



    Similar Products

    primary antibodies against aoc1  (Proteintech)
    92
    Proteintech primary antibodies against aoc1
    <t>AOC1</t> is highly expressed in gastric cancer tissues compared with normal tissues. ( A ) The red and gray boxes indicate gastric cancer and normal tissues, respectively. Data were obtained from the GEPIA website, including 408 gastric cancer samples and 211 controls. The y-axis indicated the log2-transformed gene expression levels. qPCR ( B ) and western blot ( C ) were performed to detect AOC1 expression in the tumor and paracancerous tissues of 30 patients with gastric cancerthe. *P<0.05; **P<0.01; ***P<0.001.
    Primary Antibodies Against Aoc1, supplied by Proteintech, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/primary antibodies against aoc1/product/Proteintech
    Average 92 stars, based on 1 article reviews
    primary antibodies against aoc1 - by Bioz Stars, 2026-03
    92/100 stars
    		  Buy from Supplier

    Image Search Results


    AOC1 is highly expressed in gastric cancer tissues compared with normal tissues. ( A ) The red and gray boxes indicate gastric cancer and normal tissues, respectively. Data were obtained from the GEPIA website, including 408 gastric cancer samples and 211 controls. The y-axis indicated the log2-transformed gene expression levels. qPCR ( B ) and western blot ( C ) were performed to detect AOC1 expression in the tumor and paracancerous tissues of 30 patients with gastric cancerthe. *P<0.05; **P<0.01; ***P<0.001.

    Journal: Cancer Management and Research

    Article Title: AOC1 Contributes to Tumor Progression by Promoting the AKT and EMT Pathways in Gastric Cancer

    doi: 10.2147/CMAR.S225229

    Figure Lengend Snippet: AOC1 is highly expressed in gastric cancer tissues compared with normal tissues. ( A ) The red and gray boxes indicate gastric cancer and normal tissues, respectively. Data were obtained from the GEPIA website, including 408 gastric cancer samples and 211 controls. The y-axis indicated the log2-transformed gene expression levels. qPCR ( B ) and western blot ( C ) were performed to detect AOC1 expression in the tumor and paracancerous tissues of 30 patients with gastric cancerthe. *P<0.05; **P<0.01; ***P<0.001.

    Article Snippet: Primary antibodies against AOC1 (Cat#16338-1-AP, 1:1000), GAPDH (Cat# 60004-1-Ig, 1:10,000), Bax (Cat# 50599-2-Ig, 1:6000), Bcl2 (Cat# 12789-1-AP, 1:1000), Caspase-9 (Cat# 10380-1-AP, 1:300), Caspase-3 (Cat# 19677-1-AP, 1:10,000), AKT (Cat# 60203-2-Ig, 1:5000), p-AKT (Cat# 66444-1-Ig, 1:2000), Cyclin D1 (Cat# 60186-1-Ig, 1:10,000), p70S6K (Cat# 66638-1-Ig, 1:3000), E-cadherin (Cat# 60335-1-Ig, 1:5000), N-cadherin (Cat# 66219-1-Ig, 1:5000) and SNAIL (Cat# 26183-1-AP, 1:1000) were purchased from ProteinTech (Rosemont, IL, USA).

    Techniques: Transformation Assay, Gene Expression, Western Blot, Expressing

    AOC1 expression is effectively knocked down by siRNA transfection in human gastric cancer cells. qRT-PCR assay was used to detect the interference efficiencies of siRNAs targeting AOC1 on mRNA levels in human ( A ) AGS and ( B ) MKN45 cells. ( C ) Western blot validated the effectiveness of AOC1 knockdown on protein levels. A scrambled siRNA was used as the negative control (NC). All experiments were performed in triplicate, independently. *P<0.05.

    Journal: Cancer Management and Research

    Article Title: AOC1 Contributes to Tumor Progression by Promoting the AKT and EMT Pathways in Gastric Cancer

    doi: 10.2147/CMAR.S225229

    Figure Lengend Snippet: AOC1 expression is effectively knocked down by siRNA transfection in human gastric cancer cells. qRT-PCR assay was used to detect the interference efficiencies of siRNAs targeting AOC1 on mRNA levels in human ( A ) AGS and ( B ) MKN45 cells. ( C ) Western blot validated the effectiveness of AOC1 knockdown on protein levels. A scrambled siRNA was used as the negative control (NC). All experiments were performed in triplicate, independently. *P<0.05.

    Article Snippet: Primary antibodies against AOC1 (Cat#16338-1-AP, 1:1000), GAPDH (Cat# 60004-1-Ig, 1:10,000), Bax (Cat# 50599-2-Ig, 1:6000), Bcl2 (Cat# 12789-1-AP, 1:1000), Caspase-9 (Cat# 10380-1-AP, 1:300), Caspase-3 (Cat# 19677-1-AP, 1:10,000), AKT (Cat# 60203-2-Ig, 1:5000), p-AKT (Cat# 66444-1-Ig, 1:2000), Cyclin D1 (Cat# 60186-1-Ig, 1:10,000), p70S6K (Cat# 66638-1-Ig, 1:3000), E-cadherin (Cat# 60335-1-Ig, 1:5000), N-cadherin (Cat# 66219-1-Ig, 1:5000) and SNAIL (Cat# 26183-1-AP, 1:1000) were purchased from ProteinTech (Rosemont, IL, USA).

    Techniques: Expressing, Transfection, Quantitative RT-PCR, Western Blot, Knockdown, Negative Control

    AOC1 knockdown induces growth inhibition in human gastric cancer cells. ( A ) and ( B ) After transfection with siNC or si-AOC1, the viability of AGS and MKN45 cells was detected by using CCK-8 assay. ( C ) and ( D ) Clone formation ability of AOC1 silenced gastric cancer cells was detected. All experiments were performed in triplicate. *P<0.05.

    Journal: Cancer Management and Research

    Article Title: AOC1 Contributes to Tumor Progression by Promoting the AKT and EMT Pathways in Gastric Cancer

    doi: 10.2147/CMAR.S225229

    Figure Lengend Snippet: AOC1 knockdown induces growth inhibition in human gastric cancer cells. ( A ) and ( B ) After transfection with siNC or si-AOC1, the viability of AGS and MKN45 cells was detected by using CCK-8 assay. ( C ) and ( D ) Clone formation ability of AOC1 silenced gastric cancer cells was detected. All experiments were performed in triplicate. *P<0.05.

    Article Snippet: Primary antibodies against AOC1 (Cat#16338-1-AP, 1:1000), GAPDH (Cat# 60004-1-Ig, 1:10,000), Bax (Cat# 50599-2-Ig, 1:6000), Bcl2 (Cat# 12789-1-AP, 1:1000), Caspase-9 (Cat# 10380-1-AP, 1:300), Caspase-3 (Cat# 19677-1-AP, 1:10,000), AKT (Cat# 60203-2-Ig, 1:5000), p-AKT (Cat# 66444-1-Ig, 1:2000), Cyclin D1 (Cat# 60186-1-Ig, 1:10,000), p70S6K (Cat# 66638-1-Ig, 1:3000), E-cadherin (Cat# 60335-1-Ig, 1:5000), N-cadherin (Cat# 66219-1-Ig, 1:5000) and SNAIL (Cat# 26183-1-AP, 1:1000) were purchased from ProteinTech (Rosemont, IL, USA).

    Techniques: Knockdown, Inhibition, Transfection, CCK-8 Assay

    Knockdown of AOC1 inhibits cell invasion and migration in human gastric cancer cells. ( A ) and ( C ) Transwell assays detecting the invasion and migration of human AGS and MKN45 cells. ( B ) and ( D ) Invaded and migrated cells in si-AOC1 group or NC group were counted. All experiments were performed for three repeated times. *P<0.05.

    Journal: Cancer Management and Research

    Article Title: AOC1 Contributes to Tumor Progression by Promoting the AKT and EMT Pathways in Gastric Cancer

    doi: 10.2147/CMAR.S225229

    Figure Lengend Snippet: Knockdown of AOC1 inhibits cell invasion and migration in human gastric cancer cells. ( A ) and ( C ) Transwell assays detecting the invasion and migration of human AGS and MKN45 cells. ( B ) and ( D ) Invaded and migrated cells in si-AOC1 group or NC group were counted. All experiments were performed for three repeated times. *P<0.05.

    Article Snippet: Primary antibodies against AOC1 (Cat#16338-1-AP, 1:1000), GAPDH (Cat# 60004-1-Ig, 1:10,000), Bax (Cat# 50599-2-Ig, 1:6000), Bcl2 (Cat# 12789-1-AP, 1:1000), Caspase-9 (Cat# 10380-1-AP, 1:300), Caspase-3 (Cat# 19677-1-AP, 1:10,000), AKT (Cat# 60203-2-Ig, 1:5000), p-AKT (Cat# 66444-1-Ig, 1:2000), Cyclin D1 (Cat# 60186-1-Ig, 1:10,000), p70S6K (Cat# 66638-1-Ig, 1:3000), E-cadherin (Cat# 60335-1-Ig, 1:5000), N-cadherin (Cat# 66219-1-Ig, 1:5000) and SNAIL (Cat# 26183-1-AP, 1:1000) were purchased from ProteinTech (Rosemont, IL, USA).

    Techniques: Knockdown, Migration

    Knockdown of AOC1 induces apoptosis in human gastric cancer cells. ( A ) and ( B ) The effect of AOC1 knockdown on the apoptosis of AGS cells was detected using flow cytometry. ( C ) and ( D ) The effect of AOC1 knockdown on the apoptosis of MKN45 cells was detected using flow cytometry. All experiments were performed in triplicate. *P<0.05.

    Journal: Cancer Management and Research

    Article Title: AOC1 Contributes to Tumor Progression by Promoting the AKT and EMT Pathways in Gastric Cancer

    doi: 10.2147/CMAR.S225229

    Figure Lengend Snippet: Knockdown of AOC1 induces apoptosis in human gastric cancer cells. ( A ) and ( B ) The effect of AOC1 knockdown on the apoptosis of AGS cells was detected using flow cytometry. ( C ) and ( D ) The effect of AOC1 knockdown on the apoptosis of MKN45 cells was detected using flow cytometry. All experiments were performed in triplicate. *P<0.05.

    Article Snippet: Primary antibodies against AOC1 (Cat#16338-1-AP, 1:1000), GAPDH (Cat# 60004-1-Ig, 1:10,000), Bax (Cat# 50599-2-Ig, 1:6000), Bcl2 (Cat# 12789-1-AP, 1:1000), Caspase-9 (Cat# 10380-1-AP, 1:300), Caspase-3 (Cat# 19677-1-AP, 1:10,000), AKT (Cat# 60203-2-Ig, 1:5000), p-AKT (Cat# 66444-1-Ig, 1:2000), Cyclin D1 (Cat# 60186-1-Ig, 1:10,000), p70S6K (Cat# 66638-1-Ig, 1:3000), E-cadherin (Cat# 60335-1-Ig, 1:5000), N-cadherin (Cat# 66219-1-Ig, 1:5000) and SNAIL (Cat# 26183-1-AP, 1:1000) were purchased from ProteinTech (Rosemont, IL, USA).

    Techniques: Knockdown, Flow Cytometry

    Knockdown of AOC1 induces activation of the mitochondrial apoptosis pathway, and also inhibits the AKT signaling pathway and EMT process. ( A and B ) The key members of the mitochondrial apoptosis pathway, including Bax, Bcl2, Caspase-9, and Caspase-3, were detected by Western blot. ( C and D ) AKT signaling pathway members, including AKT, p-AKT, Cyclin D1, and p70S6K, were detected by Western blot. ( E and F ) EMT-related proteins, including E-cadherin, N-cadherin, SNAIL and Slug, were detected by Western blot. All experiments were performed in triplicate. *P<0.05.

    Journal: Cancer Management and Research

    Article Title: AOC1 Contributes to Tumor Progression by Promoting the AKT and EMT Pathways in Gastric Cancer

    doi: 10.2147/CMAR.S225229

    Figure Lengend Snippet: Knockdown of AOC1 induces activation of the mitochondrial apoptosis pathway, and also inhibits the AKT signaling pathway and EMT process. ( A and B ) The key members of the mitochondrial apoptosis pathway, including Bax, Bcl2, Caspase-9, and Caspase-3, were detected by Western blot. ( C and D ) AKT signaling pathway members, including AKT, p-AKT, Cyclin D1, and p70S6K, were detected by Western blot. ( E and F ) EMT-related proteins, including E-cadherin, N-cadherin, SNAIL and Slug, were detected by Western blot. All experiments were performed in triplicate. *P<0.05.

    Article Snippet: Primary antibodies against AOC1 (Cat#16338-1-AP, 1:1000), GAPDH (Cat# 60004-1-Ig, 1:10,000), Bax (Cat# 50599-2-Ig, 1:6000), Bcl2 (Cat# 12789-1-AP, 1:1000), Caspase-9 (Cat# 10380-1-AP, 1:300), Caspase-3 (Cat# 19677-1-AP, 1:10,000), AKT (Cat# 60203-2-Ig, 1:5000), p-AKT (Cat# 66444-1-Ig, 1:2000), Cyclin D1 (Cat# 60186-1-Ig, 1:10,000), p70S6K (Cat# 66638-1-Ig, 1:3000), E-cadherin (Cat# 60335-1-Ig, 1:5000), N-cadherin (Cat# 66219-1-Ig, 1:5000) and SNAIL (Cat# 26183-1-AP, 1:1000) were purchased from ProteinTech (Rosemont, IL, USA).

    Techniques: Knockdown, Activation Assay, Western Blot

    IGF-1 blocked the inhibition of cell proliferation, migration and invasion caused by AOC1 knockdown. AOC1 low expressing cells were treated with IGF-1, an agonist of the AKT pathway. ( A ) CCK8 was performed to detected the proliferation of each group cells. ( B ) Transwell was performed to detected the migration and invasion of each group cells. *P<0.05 vs. NC group; #P<0.05 vs. si-AOC1 group.

    Journal: Cancer Management and Research

    Article Title: AOC1 Contributes to Tumor Progression by Promoting the AKT and EMT Pathways in Gastric Cancer

    doi: 10.2147/CMAR.S225229

    Figure Lengend Snippet: IGF-1 blocked the inhibition of cell proliferation, migration and invasion caused by AOC1 knockdown. AOC1 low expressing cells were treated with IGF-1, an agonist of the AKT pathway. ( A ) CCK8 was performed to detected the proliferation of each group cells. ( B ) Transwell was performed to detected the migration and invasion of each group cells. *P<0.05 vs. NC group; #P<0.05 vs. si-AOC1 group.

    Article Snippet: Primary antibodies against AOC1 (Cat#16338-1-AP, 1:1000), GAPDH (Cat# 60004-1-Ig, 1:10,000), Bax (Cat# 50599-2-Ig, 1:6000), Bcl2 (Cat# 12789-1-AP, 1:1000), Caspase-9 (Cat# 10380-1-AP, 1:300), Caspase-3 (Cat# 19677-1-AP, 1:10,000), AKT (Cat# 60203-2-Ig, 1:5000), p-AKT (Cat# 66444-1-Ig, 1:2000), Cyclin D1 (Cat# 60186-1-Ig, 1:10,000), p70S6K (Cat# 66638-1-Ig, 1:3000), E-cadherin (Cat# 60335-1-Ig, 1:5000), N-cadherin (Cat# 66219-1-Ig, 1:5000) and SNAIL (Cat# 26183-1-AP, 1:1000) were purchased from ProteinTech (Rosemont, IL, USA).

    Techniques: Inhibition, Migration, Knockdown, Expressing